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Literature review on lateral phoria

A case of posterior communicating artery infundibulum has been documented causing recurrent ipsilateral third cranial nerve palsy and headache that masqueraded as ophthalmoplegic mraine. Weber syndrome results from a slhtly more ventral lesion at the level of the third cranial nerve fascicles in the mid brain, with involvement of the cerebral peduncle giving rise to contralateral hemiplegia or hemiparesis along with ipsilateral third cranial nerve palsy. Ophthalmoplegic mraine presents in childhood with recurring bouts of unilateral headache and ipsilateral third cranial nerve palsy that can last several weeks at a time.

Literature review on lateral phoria

Literature review on lateral phoria

Most of the lesions causing nuclear, third cranial nerve palsy are from dorsal midbrain infarction. Since the most common lesion to affect the third cranial nerve in the subarachnoid space is aneurysm, the sns and symptoms of subarachnoid hemorrhage, including sudden severe headache, stiff neck, and loss of consciousness, may be present.

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  • The findings of third cranial nerve palsy tend to occur in isolation from lesions in this location.


    Literature review on lateral phoria

    Literature review on lateral phoria

    Literature review on lateral phoria

    The third cranial nerve is more susceptible to compression against the interclinoid laments above and the petroclinoid lament below than are the other cranial nerves in the cavernous sinus. Subarachnoid portion The fascicles of the third cranial nerve exit the mid brain through the medial aspect of the cerebral peduncles and are not near any of the other cranial nerves at this point.

    Literature review on lateral phoria

    Convergence insufficiency (CI) patients before vergence therapy were compared to: (1) the same patients after vergence therapy; and (2) binocularly normal controls (BNC). PREVENT UNEMPLOYMENT ESSAY Fascicular midbrain portion Benedikt syndrome of the upper mid brain includes third cranial nerve palsy on the side of the lesion, ipsilateral flapping hand tremor (rubral tremor from red nucleus involvement), and ataxia.


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